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Transcript

so bad cholesterol LDL is not

cholesterol and is not bad

okay so the first point I want to bring

up just to try to make this really

simple is you have these little

factories in your cells okay and you

have certain factories that make

proteins okay and basically what they do

is they take the building blocks of

protein which are amino acids and they

start connecting them in certain shapes

to form different proteins and that’s

what you know skin hair nails certain

hormones you know transmitters they’re

just different shapes of assembled amino

acids okay so one factory makes protein

the other one makes lipids certain fats

that make up your body you got fat in

your skin yes fat in your brain and your

nervous system okay then we have

steroids that make certain hormones like

estrogen progesterone testosterone

cortisol things like that and then we

have a detox function as well okay so if

you drink alcohol for example your body

has to break that down well it’s this

factory that breaks it down and the name

of this factory is called rough

endoplasmic reticulum and this factory

is called smooth endoplasmic reticulum

not that you need to know that but this

is where it starts okay and this has to

be sent to a dispatch center like a post

office okay so this post office packages

proteins for sending and being secreted

through the body so it’s very similar to

the post office where they have to

package it and put a label on it okay

with postage so it could be sent out

that’s called the Golgi body so one of

the things that these two mechanisms

make our cargo boats because the fat

needs to be delivered through the blood

and it doesn’t dissolve well so oil and

water don’t mix well so it has to be

packaged into a protein so this is what

a lipoprotein is it’s a protein shell

that holds fat

so it could be transported called a

cargo boat now let’s go to the liver

right here we have the liver when you’re

not eating makes these cargo boats okay

and inside the liver you have these two

mechanisms going on right here

and so these lipoproteins are made and

one of the lipoproteins is called VLDL

very low density lipoprotein basically

you have certain percentages of

cholesterol triglycerides protein and

other fats and all the fat soluble

vitamins you have vitamin A II D k1 k2

okay so the purpose of a V LDL is to

send out this this cargo and unload

these materials okay and these vitamins

so it provides the fuel to the cell like

in triglycerides it provides material

for the cell walls it provides material

to build bile make vitamin D certain

hormones and of course the antioxidants

as well okay so when this unloads its

cargo it turns into a LDL low-density

lipoprotein now what’s the difference

between this and this well we just have

different percentages of materials so

we’re going from 10% cholesterol to 26%

cholesterol we’re going up because we’re

lowering the triglycerides so the ratio

is going to be a little bit higher that

make sense cell so look at the 70%

triglycerides in this but now then the

10 so it dumps a lot of triglyceride for

the fuel to feed the cells and 10%

protein 25% protein okay 10 percent

other fats and 15 percent other fats so

basically just see the ratios are

changed and that’s really the difference

now people associated this with bad

cholesterol because this happens to be

in the location we have a clogged artery

well guess what it’s at the location of

the crime scene but it’s there to help

heal it okay

to provide a band-aid to bring the

cholesterol to help heal it LDL is only

dangerous if you have oxidized

cholesterol okay now to understand this

a little bit more I downloaded a very

important research article evidence that

acute insulin administration enhances

LDL cholesterol susceptibility to

oxidation and healthy humans now

basically I’m just going to give you the

conclusion okay I’m gonna read this to

you in conclusion insulin at

physiological doses that means greater

amounts of insulin is associated with

increase LDL parry oxidation basically

it’s oxidized cholesterol right that’s

what we’re talking about independent of

the presence of hyperglycemia and

basically insulin can oxidize

cholesterol regardless of how high your

blood sugars are okay now why do they

say that because of this next point

right here this result supports the

possibility that insulin resistance okay

may be implicated in the genesis of

early atherosclerosis and/or the

progression of atherosclerosis which is

hardening of the arteries through the

attendant hyperinsulinemia what does

that mean it means that you could have

oxidized cholesterol if the insulin is

high but it’s not necessarily coming

from eating sugar necessarily it’s

coming because you have insulin

resistance and then of course originally

came from eating sugar but insulin

resistance itself produces a lot more

insulin so it’s a kind of a feedback

mechanism that you’re producing about

five to seven times more insulin than a

normal person and that can create the

oxidation of cholesterol so we have two

things that can create bad LDL one is

high sugar and the other is high insulin

okay so I just wanted to point that out

and I’ll put a link down below if you

wanted to read this

you don’t have to worry about having a

higher LDL if you’re on keto as long as

your Sugar’s down and your insulin is

lower by doing healthy keto and

intermittent fasting over a period of

time what’s going to happen is your

insulin resistance is going to heal and

the cholesterol is eventually going to

come down alright so I just wanted to

make this as simple as possible I hope

you understand a little bit better you

don’t have to worry about this it’s not

bad it’s a boat

that’s there for a reason it’s

transporting fuel it’s trying to help

you the bottom line is you just need to

do things to keep your blood sugars and

your insulin in a normal range thanks

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